Allow me to present indisputable evidence on the critical role testosterone plays in physical and mental strength.
Testosterone, the potent hormone predominantly produced in the testes of males and is essential for a wide array of physiological and psychological functions.
Beware the consequences of low testosterone, as they include:
- Reduced muscle mass and strength: Testosterone is the lifeblood for muscle development and maintenance. Low levels of this hormone can lead to weakened muscle mass, diminished strength, and feeble endurance.
- Increased body fat: Testosterone stands guard over body fat distribution. When levels are low, individuals may fall victim to an increase in body fat, particularly around the abdomen.
- Reduced bone density: Testosterone is the stalwart defender of bone density. Low testosterone levels can surrender to weaker bones and an increased risk of fractures.
- Lower energy levels: Low testosterone acts as a saboteur, resulting in fatigue, reduced stamina, and overall diminished energy levels.
- Decreased libido: Testosterone is the linchpin for maintaining sexual desire and function. Low levels can extinguish the fire of libido and trigger erectile dysfunction in men.
- Mood disturbances: Low testosterone levels can unleash emotional chaos, leading to mood swings, irritability, depression, and anxiety.
- Cognitive decline: Testosterone is the vital force for maintaining cognitive function, and low levels may summon the fog of memory and concentration issues.
Now, with unwavering confidence, I present the evidence supporting the importance of testosterone for physical and mental strength:
- Muscle growth and strength: Testosterone fuels the production of muscle proteins, which form the building blocks of muscle tissue. This process, known as protein synthesis, leads to increased muscle size and strength (1).
- Bone density: Testosterone fortifies bone mineral density by stimulating bone formation and inhibiting bone resorption (2).
- Fat metabolism: Testosterone deftly orchestrates the regulation of adipose tissue, influencing the distribution of body fat and promoting the breakdown of fat for energy (3).
- Energy levels: Testosterone drives various metabolic processes that provide energy for physical and mental activities. Adequate levels of testosterone are the key to maintaining optimal energy levels (4).
- Libido and sexual function: Testosterone is the master regulator of sexual desire and function, ensuring healthy libido levels and peak erectile function in men (5).
- Mood regulation: Testosterone has a profound impact on mood by influencing neurotransmitters in the brain, such as serotonin and dopamine (6).
- Cognitive function: Testosterone elevates cognitive performance in various domains, including memory, attention, and spatial abilities (7).
In a nutshell, testosterone is undeniably critical for physical and mental strength due to its significant role in muscle growth, bone density, fat metabolism, energy production, sexual function, mood regulation, and cognitive performance.
Low levels of testosterone can spawn a host of health issues, emphasizing the importance of maintaining optimal levels for overall well-being.
So why do Dr’s brush this of as no big deal? Because it’s kind of a BIG deal.
Maybe this video will shine the light on why
Low Fasting Insulin: Can Be The Key to Boosting Testosterone
In this video, we explore the fascinating connection, delve into the research, and offer practical tips to getting insulin levels optimised even if you are prediabetic.
Because the research is clear, elevated insulin can cause a huge up-regulation of the aromatase enzyme that converts testosterone into estrogen.
The connection between elevated insulin levels and the aromatase enzyme has been well-established through scientific research. Insulin, a hormone produced by the pancreas, is responsible for regulating glucose levels in the bloodstream.
When insulin levels are high, it can lead to a significant increase in the activity of the aromatase enzyme. The aromatase enzyme plays a crucial role in the conversion of androgens, such as testosterone, into estrogens, like estradiol.
When there is an up-regulation of the aromatase enzyme due to high insulin levels, it can result in the excessive conversion of testosterone into estrogen.
This hormonal imbalance can have several negative consequences for both men and women.
In men, high estrogen levels can lead to a reduction in muscle mass, an increase in body fat, gynecomastia (enlarged breast tissue), reduced libido, and even erectile dysfunction.
Additionally, when testosterone is converted into estrogen, it leads to a decrease in the overall testosterone levels, which can further exacerbate these symptoms and impact overall health, mood, and well-being. So the key to higher testosterone levels starts with optimal fasting insulin.
- 00:00 – Starting with inflammation and muscle building
- 01:45 – How elevated insulin lowers testosterone and boosts estrogen levels up to 15-fold
- 04:55 – What are the symptons of low testosterone?
- 06:00 – How your ball sack makes testosteorne
- 08:05 – The consequences of too much insulin
- 09:50 – How elevated ferritin can significantly lower testosterone
- 11:00 – How to lower estrogen levels
- 12:15 – homocysteine optimal levels and protein
- 14:20 – How elevated insulin lowers growth hormone
- 16:45 – Estrogen, testosterone and mindset
- 19:00 – How to lower insulin and blood glucose
- 24:55 – Summary and final thoughts
If you’re interested in getting back in the mental and physical game, book a free optimisation call to get your energy and testosterone back below:
The game is utterly rigged against us, ruthlessly sabotaging our testosterone levels.
They deliberately avoid measuring fasting insulin because they fear us discovering how to reclaim our testosterone. Instead, they’d prefer big pharma to profit from our weakened state.
Let’s face it: This entire system is waging a brutal hormonal war on us, and it’s high time we rise and retaliate.
Why? Because men with high testosterone are a menace to the powers that be.
We’re unapologetically self-reliant, steadfast, and refuse to meekly submit to their oppressive norms.
We defend our loved ones with ferocity and won’t hesitate to ensure their prosperity.
It’s no surprise they’re hell-bent on tearing us down.
Understand this: You’re not broken, you’re not feeble, and you’re not isolated. Your once-powerful hormones have been tampered with.
Keep in mind, it’s not just your mindset that shapes your day; it’s the undeniable influence of those hormone levels.
- Vingren, J. L., Kraemer, W. J., Ratamess, N. A., Anderson, J. M., Volek, J. S., & Maresh, C. M. (2010). Testosterone physiology in resistance exercise and training. Sports Medicine, 40(12), 1037-1053.
- Orwoll, E., & Klein, R. F. (1995). Osteoporosis in men. Endocrine Reviews, 16(1), 87-116.
- Stanworth, R. D., & Jones, T. H. (2008). Testosterone for the aging male; current evidence and recommended practice. Clinical Interventions in Aging, 3(1), 25-44.
- Bhasin, S., Brito, J. P., Cunningham, G. R., Hayes, F. J., Hodis, H. N., Matsumoto, A. M., … & Yialamas, M. A. (2018). Testosterone therapy in men with hypogonadism: an Endocrine Society clinical practice guideline. The Journal of Clinical Endocrinology & Metabolism, 103(5), 1715-1744.
- Corona, G., Rastrelli, G., Morgentaler, A., Sforza, A., Mannucci, E., & Maggi, M. (2016). Meta-analysis of results of testosterone therapy on sexual function based on international index of erectile function scores. European Urology, 72(6), 1000-1011.
- Wang, C., Cunningham, G., Dobs, A., Iranmanesh, A., Matsumoto, A. M., Snyder, P. J., … & Swerdloff, R. S. (2009). Long-term testosterone gel (AndroGel) treatment maintains beneficial effects on sexual function and mood, lean and fat mass, and bone mineral density in hypogonadal men. The Journal of Clinical Endocrinology & Metabolism, 94(5), 1587-1597.
- Cherrier, M. M., Asthana, S., Plymate, S., Baker, L., Matsumoto, A. M., Peskind, E., … & Craft, S. (2001). Testosterone supplementation improves spatial and verbal memory in healthy older men. Neurology, 57(1), 80-88.